Inflammation is so cross disciplinary that to draw interest and focus on journals conferences which may need to be quite on specific areas. A better understanding of the different mechanisms and interaction between inflammatory networks is needed and more in disciplinary communication is required for discussion of various important aspects of the inflammation. A separation of inflammation from immunology may not be beneficial although an improvement in the profile of “inflammation” within the immunology community may be productive.
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Acute inflammation begins within seconds to minutes following the injury of tissues. The damage may be purely physical, or it may involve the activation of an immune response. Sometimes pus forms at the site of acute inflammation, especially if a foreign body is present to continually aggravate the tissue.
Related Journals of Acute Inflammation
Archives of Inflammation, Inflammatory Bowel Diseases & Disorders, Immunome Research, Immunogenetics: Open Access, ImmunologicalTechniques in Infectious Diseases, Inflammation, Inflammation Research, Inflammation and Allergy - Drug Targets, Pediatric Allergy & Immunology, Pediatric Asthma, Allergy and Immunology.
Allergic inflammation is due to a complex interaction between several inflammatory cells, including mast cells, basophils, lymphocytes, dendritic cells, eosinophil, and sometimes neutrophils. These cells produce multiple inflammatory mediators, including lipids, purines, cytokines, chemokine, and reactive oxygen species.
Related Journals of Allergic Inflammation
Archives of Inflammation, Immunome Research, Immunooncology, Immunotherapy: Open Access, Infectious Diseases & Therapy, Recent Patents on Inflammation & Allergy Drug Discovery, Current Drug Targets - Inflammation & Allergy, Current Opinion in Allergy and Clinical Immunology, Current Topics in Microbiology and Immunology, Expert Review of Clinical Immunology.
Ocular Inflammation is inflammation of the uvea, the middle layer of your eye. The eye is shaped much like a tennis ball, with three different layers of tissue surrounding a central gel-filled cavity. The uvea contains many blood vessels, the veins and arteries that carry blood flow to the eye. Since it nourishes many important parts of the eye (such as the retina), inflammation of the uvea can damage your sight. Ocular inflammation may be the result of a wide variety of causes, including infection and inflammatory disorders (see below). Some conditions may affect other parts of the body. In many cases however, despite thorough investigations, the cause remain unknown.
Related Journals of Ocular Inflammation
Archives of Inflammation, Infectious Diseases and Diagnosis , Infectious Diseases and Treatment , Inflammatory Bowel Diseases & Disorders, Innate Immunity & Immunological Disorders , Ocular Immunology and Inflammation, OncoImmunology, Journal of Ophthalmic Inflammation and Infection, International Journal of Inflammation, Mucosal Immunology.
in some people, the inflammatory response is triggered inappropriately or never fully shuts off, potentially resulting in a state of chronic inflammation. Exactly how biochemical processes cause chronic inflammation is not yet understood, but we do know that women and older people suffer more inflammatory illnesses. Chronic inflammatory demyelinating polyneuropathy is the most common chronic neuropathy caused by an abnormal immune response. CIDP occurs when the immune system attacks the myelin cover of the nerves.
Related Journals of Chronic Inflammatiion
Archives of Inflammation, Interdisciplinary Journal of Microinflammation, Immunobiology, Clinical & Cellular Immunology, Molecular Immunology, European Journal of Inflammation, Inflammation and Regeneration, Inflammation Research, Journal of Inflammation, Immunobiology.
Mucosal inflammation typically refers to swelling or irritation of the mucus membranes. These are areas of the body which produce mucus in an effort to filter out bacteria, viruses, and other invaders. This includes the nasal cavities, mouth, throat, eyes, vagina, lungs, and intestines. Inflammation may occur if bacteria or viruses cause an infection, if the area is irritated by allergens or other foreign bodies, or due to fungal infection.
Related Journals of Mucosal Inflammation
Archives of Inflammation, Inflammatory Bowel Diseases & Disorders, Molecular Immunology, Immunome Research, Infectious Diseases and Treatment, Mucosal Immunology, Immunity and Ageing, Inflammation and Cancer, Modern Research in Inflammation, Immunity, Inflammation and Disease, The Internet Journal of Asthma, Allergy and Immunology.
The endogenous compounds that mediate inflammation (autacoids) and related exogenous compounds including the synthetic prostaglandins. Epithelial cells are the first point of host contact for invasive intestinal pathogens and may initiate mucosal inflammatory responses via production of Proinflammatory cytokines and mediators.
Related Journals of Inflammatory Mediators
Archives of Inflammation, Infectious Diseases & Therapy, Infectious Diseases and Diagnosis, Infectious Diseases and Treatment, Inflammatory Bowel Diseases & Disorders, Mediators of Inflammation, International Journal of Interferon Cytokine and Mediator Research, Journal of Allergy, International Archives of Allergy and Immunology, International Immunopharmacology.
The eosinophil is well recognized as a central effector cell in the inflamed asthmatic airway. Eosinophils release toxic basic proteins and lipid mediators such as cysteinyl-leukotrienes that cause bronchial epithelial damage and airflow obstruction. Eosinophil-selective cytokines and chemokine including interleukin (IL)-5, eotaxin and RANTES may represent targets for novel asthma therapies.
Related Journals of Eosinophilic Inflammation
Archives of inflammation, Immunogenetics: Open Access, Infectious Diseases & Therapy, Immunome Research, Immunome Research, Immunology, Immunology and Allergy Clinics of North America, Journal of Allergy and Asthma, Immunopharmacology and Immunotoxicology, Indian Journal of Allergy Asthma and Immunology.
The granulomatous inflammatory response is a special type of chronic inflammation characterized by often focal collections of macrophages, epithelioid cells and multinucleated giant cells. Granulomas are masses of immune cells that form at sites of infection or inflammation. These severe infections can include skin or bone infections and abscesses in internal organs (such as the lungs, liver or brain).
Related Journals of Fibrinous Inflammation
Archives of Inflammation, Immunome Research, Immunogenetics: Open Access , Immunological Techniques in Infectious Diseases, Immunome Research , Recent Patents on Inflammation & Allergy Drug Discovery, Review of Allergy and Clinical Immunology, International Review of Allergology and Clinical Immunology, Journal of Allergy & Therapy, International Journal of Immunopharmacology
An inflammatory reaction in which the distinguishing feature is an increase in the number of tissue cells, especially the reticulo endothelial macrophages, rather than of cells exuded from blood vessels. In addition, exudates of various types are likely to be observed in granulomas and other forms of proliferative inflammation, but the latter may occur without an exudate being formed (as in certain infections caused by virus).
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Archives of Inflammation, Inflammatory Bowel Diseases & Disorders , Innate Immunity & Immunological Disorders , Interdisciplinary Journal of Microinflammation, Immunobiology, Clinical and Experimental Allergy Reviews, Clinical and Experimental Immunology, Clinical and Molecular Allergy, Clinical and Translational Allergy, Clinical Reviews in Allergy and Immunology.
It is also called as lymphocytic inflammation. Suppurative is a term used to describe a disease or condition in which a purulent exudate (pus) is formed and discharged. Examples of such condition include appendicitis, arthritis, cerebritis, choroiditis, encephalitis, endophthalmitis, gastritis, gingivitis, hepatitis, hyalitis, mastitis, nephritis, pancreatitis, periodontitis, and phlebitis. Inflammation accompanied by pus formation is referred to as suppurating inflammation. In contrast, an inflammation not accompanied or characterized by suppuration is called nonsuppurative inflammation.
Related Journals of Suppurative Inflammation
Archives of inflammation, Immunogenetics: Open Access, Infectious Diseases & Therapy, Immunome Research, Immunome Research, Clinical and Experimental Allergy Reviews, Clinical and Experimental Immunology, Clinical and Molecular Allergy, Clinical and Translational Allergy, Clinical Reviews in Allergy and Immunology.
An inflammatory process in which there is infiltration of polymorphonuclear cells into a more chronic area of inflammation characterized by mononuclear cells, macrophages, lymphocytes and possibly plasma cells. Actinomyces sp. is gram-positive, acid-fast–negative filamentous bacteria that cause pyogranulomatous infections in dogs, cats, cattle, goats, swine, horses, foxes and human beings.
Related Journals of Pyogranulomatous Inflammation
Archives of Inflammation, Infectious Diseases and Diagnosis, Infectious Diseases and Treatment, Inflammatory Bowel Diseases & Disorders, Innate Immunity & Immunological Disorders, Advances in Immunology, the Open Inflammation Journal, Complement and Inflammation, European Journal of Rheumatology & Inflammation, Progress in Inflammation Research, Clinical and Applied Immunology Reviews.
The inflammatory reaction to injury begins with a dilation of blood vessels and an increased blood flow. This is accompanied by an increased permeability of vessel walls, which leads to the escape of a protein-rich fluid into the interstitial spaces. The fluid is an exudate, forced out of the vessel by increased pressure and leakiness. Serous, Fibrinous and Suppurative inflammation are all manifestations of a spectrum of increased vascular permeability caused by inflammation.
Related Journals of Serous Inflammation
Archives of inflammation, Immunological Techniques in Infectious Diseases, Immunome Research, Immunooncology, Immunotherapy: Open Access, Immunological Investigations, Chemical Immunology and Allergy, Allergy and Clinical Immunology, International Allergy Asthma & Clinical Immunology, Asian Pacific Journal of Allergy & Immunology, Annals of Allergy Asthma & Immunology.
An inflammation not accompanied or characterized by suppuration is called nonsuppurative inflammation. Non-Suppurative inflammations are divided into 5 types that is Catarrhal inflammation, Fibrinous inflammation which may be serous or serofibrinous, Membranous inflammation, Hemorrhagic inflammation, Allergic inflammation.
Related Journals of Nonsuppurative Inflammation
Archives of Inflammation, Immunogenetics: Open Access, Immunological Techniques in Infectious Diseases, Immunome Research, Immunooncology, Trends in Immunology, International Immunopharmacology, Journal of Autoimmunity, Journal of Immunological Methods, The Open Inflammation Journal.
Chemical mediators of the inflammatory process include a variety of substances originating in the plasma and the cells of uninjured tissue, and possibly from the damaged tissue. The major kinds of mediators are (1) vasoactive amines, such as histamine and serotonin; (2) plasma endopeptidases that comprise three interrelated systems, the kinin system that produces bradykinin, the complement system that produces proteins that interact with antigen--antibody complexes and mediate immunologic injury and inflammation, and the clotting system that increases vascular permeability and chemotactic activity for the leukocytes.
Related Journals of Proinflammatory Mediators
Archives of inflammation, Immunogenetics: Open Access, Infectious Diseases & Therapy, Immunome Research, Immunome Research, Journal of Interferon & Cytokine Research, Cytokine, Inflammation and Regeneration, Inflammation Research, Inflammation Research.
Ulcerative colitis (UC) is a chronic inflammatory and ulcerative disease arising in the colonic mucosa, characterized most often by bloody diarrhea. Extra intestinal symptoms, particularly arthritis, may occur. Long-term risk of colon cancer is high. Diagnosis is by colonoscopy. Treatment is with 5-aminosalicylic acid, corticosteroids, immunomodulators, anticytokines, antibiotics, and occasionally surgery.
Related Journals of Ulcerative Inflammation
Archives of inflammation, Immunogenetics: Open Access, Infectious Diseases & Therapy, Immunome Research, Immunome Research, Cytokine & Growth Factor Reviews, Current opinion on Immunology, Developmental & Comparative Immunology, Human Immunology, Immunology Letters.
Author(s): Alicia Galindo-Ferreiro, Sahar M Elkhamary, Hind Alkatan, Azza Maktabi, Alberto Galvez-Ruizand Silvana Schellini
Author(s): Havva Ozge Keseroglu, Necip Enis Kaya, Aysun Gokce, Muzeyyen Gonul
Author(s): Yuji Shimizu, Mio Nakazato, Shimpei Sato, Mako Nagayoshi, Koichiro Kadota, Yuko Noguchi, Jun Koyamatsu, Hirotomo Yamanashi, Kazuhiko Arima and Takahiro Maeda
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