Archives of Inflammation

About Archives of Inflammation

Inflammation is so cross disciplinary that to draw interest and focus on journals conferences which may need to be quite on specific areas. A better understanding of the different mechanisms and interaction between inflammatory networks is needed and more in disciplinary communication is required for discussion of various important aspects of the inflammation. A separation of inflammation from immunology may not be beneficial although an improvement in the profile of “inflammation” within the immunology community may be productive.

Archives of Inflammation welcomes research submissions on all aspects of inflammation which is an open access, peer-reviewed online journal. The intention is that the journal should reflect both the experimental and clinical aspects of inflammation. We will facilitate article submission, rapid article quality assurance through 'peer review', article formatting and processing to a final product which will allow high visibility, impact and provoke debate.

Submit manuscript at www.editorialmanager.com/imedpubjournals/ or

send as an e-mail attachment to the Editorial Office at inflammation@imedpub.com

 

 

Acute Inflammation

Acute inflammation begins within seconds to minutes following the injury of tissues. The damage may be purely physical, or it may involve the activation of an immune response. Sometimes pus forms at the site of acute inflammation, especially if a foreign body is present to continually aggravate the tissue.

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Archives of Inflammation, Inflammatory Bowel Diseases & Disorders, Immunome Research, Immunogenetics: Open Access , ImmunologicalTechniques in Infectious Diseases, Inflammation, Inflammation Research, Inflammation and Allergy - Drug Targets, Pediatric Allergy & Immunology, Pediatric Asthma, Allergy and Immunology

 

Allergic Inflammation

Allergic inflammation is due to a complex interaction between several inflammatory cells, including mast cells, basophils, lymphocytes, dendritic cells, eosinophil, and sometimes neutrophils. These cells produce multiple inflammatory mediators, including lipids, purines, cytokines, chemokine, and reactive oxygen species

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Archives of Inflammation, Immunome Research, Immunooncology, Immunotherapy: Open Access, Infectious Diseases & Therapy, Recent Patents on Inflammation & Allergy Drug Discovery, Current Drug Targets - Inflammation & Allergy, Current Opinion in Allergy and Clinical Immunology, Current Topics in Microbiology and Immunology, Expert Review of Clinical Immunology

 

 

Ocular Inflammation

Ocular Inflammation is inflammation of the uvea, the middle layer of your eye. The eye is shaped much like a tennis ball, with three different layers of tissue surrounding a central gel-filled cavity. The uvea contains many blood vessels, the veins and arteries that carry blood flow to the eye. Since it nourishes many important parts of the eye (such as the retina), inflammation of the uvea can damage your sight. Ocular inflammation may be the result of a wide variety of causes, including infection and inflammatory disorders (see below). Some conditions may affect other parts of the body. In many cases however, despite thorough investigations, the cause remain unknown.

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Archives of Inflammation, Infectious Diseases and Diagnosis , Infectious Diseases and Treatment , Inflammatory Bowel Diseases & Disorders, Innate Immunity & Immunological Disorders , Ocular Immunology and Inflammation, OncoImmunology, Journal of Ophthalmic Inflammation and Infection, International Journal of Inflammation, Mucosal Immunology,

Chronic Inflammation

in some people, the inflammatory response is triggered inappropriately or never fully shuts off, potentially resulting in a state of chronic inflammation. Exactly how biochemical processes cause chronic inflammation is not yet understood, but we do know that women and older people suffer more inflammatory illnesses. Chronic inflammatory demyelinating polyneuropathy is the most common chronic neuropathy caused by an abnormal immune response. CIDP occurs when the immune system attacks the myelin cover of the nerves.

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Archives of Inflammation, Interdisciplinary Journal of Microinflammation, Immunobiology, Clinical & Cellular Immunology, Molecular Immunology, European Journal of Inflammation, Inflammation and Regeneration, Inflammation Research, Journal of Inflammation, Immunobiology

 

Mucosal inflammation

Mucosal inflammation typically refers to swelling or irritation of the mucus membranes. These are areas of the body which produce mucus in an effort to filter out bacteria, viruses, and other invaders. This includes the nasal cavities, mouth, throat, eyes, vagina, lungs, and intestines. Inflammation may occur if bacteria or viruses cause an infection, if the area is irritated by allergens or other foreign bodies, or due to fungal infection

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Archives of Inflammation, Inflammatory Bowel Diseases & Disorders, Molecular Immunology, Immunome Research, Infectious Diseases and Treatment, Mucosal Immunology, Immunity and Ageing, Inflammation and Cancer, Modern Research in Inflammation, Immunity, Inflammation and Disease, The Internet Journal of Asthma, Allergy and Immunology.

Inflammatory Mediators

The endogenous compounds that mediate inflammation (autacoids) and related exogenous compounds including the synthetic prostaglandins. Epithelial cells are the first point of host contact for invasive intestinal pathogens and may initiate mucosal inflammatory responses via production of Proinflammatory cytokines and mediators.

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Eosinophilic Inflammation

The eosinophil is well recognized as a central effector cell in the inflamed asthmatic airway. Eosinophils release toxic basic proteins and lipid mediators such as cysteinyl-leukotrienes that cause bronchial epithelial damage and airflow obstruction. Eosinophil-selective cytokines and chemokine including interleukin (IL)-5, eotaxin and RANTES may represent targets for novel asthma therapies.

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Fibrinous Inflammation

The granulomatous inflammatory response is a special type of chronic inflammation characterized by often focal collections of macrophages, epithelioid cells and multinucleated giant cells. Granulomas are masses of immune cells that form at sites of infection or inflammation. These severe infections can include skin or bone infections and abscesses in internal organs (such as the lungs, liver or brain).

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Hyperplastic Inflammation

An inflammatory reaction in which the distinguishing feature is an increase in the number of tissue cells, especially the reticulo endothelial macrophages, rather than of cells exuded from blood vessels. In addition, exudates of various types are likely to be observed in granulomas and other forms of proliferative inflammation, but the latter may occur without an exudate being formed (as in certain infections caused by virus).

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Suppurative Inflammation

It is also called as lymphocytic inflammation. Suppurative is a term used to describe a disease or condition in which a purulent exudate (pus) is formed and discharged. Examples of such condition include appendicitis, arthritis, cerebritis, choroiditis, encephalitis, endophthalmitis, gastritis, gingivitis, hepatitis, hyalitis, mastitis, nephritis, pancreatitis, periodontitis, and phlebitis. Inflammation accompanied by pus formation is referred to as suppurating inflammation. In contrast, an inflammation not accompanied or characterized by suppuration is called nonsuppurative inflammation.

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Archives of inflammation, Immunogenetics: Open Access, Infectious Diseases & Therapy, Immunome Research, Immunome Research, Clinical and Experimental Allergy Reviews, Clinical and Experimental Immunology, Clinical and Molecular Allergy, Clinical and Translational Allergy, Clinical Reviews in Allergy and Immunology


Pyogranulomatous Inflammation

An inflammatory process in which there is infiltration of polymorphonuclear cells into a more chronic area of inflammation characterized by mononuclear cells, macrophages, lymphocytes and possibly plasma cells. Actinomyces sp. is gram-positive, acid-fast–negative filamentous bacteria that cause pyogranulomatous infections in dogs, cats, cattle, goats, swine, horses, foxes and human beings.

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Archives of Inflammation, Infectious Diseases and Diagnosis, Infectious Diseases and Treatment, Inflammatory Bowel Diseases & Disorders, Innate Immunity & Immunological Disorders, Advances in Immunology, the Open Inflammation Journal, Complement and Inflammation, European Journal of Rheumatology & Inflammation, Progress in Inflammation Research, Clinical and Applied Immunology Reviews

Serous Inflammation

The inflammatory reaction to injury begins with a dilation of blood vessels and an increased blood flow. This is accompanied by an increased permeability of vessel walls, which leads to the escape of a protein-rich fluid into the interstitial spaces. The fluid is an exudate, forced out of the vessel by increased pressure and leakiness. Serous, Fibrinous and Suppurative inflammation are all manifestations of a spectrum of increased vascular permeability caused by inflammation.

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Nonsuppurative Inflammation

An inflammation not accompanied or characterized by suppuration is called nonsuppurative inflammation. Non-Suppurative inflammations are divided into 5 types that is Catarrhal inflammation, Fibrinous inflammation which may be serous or serofibrinous, Membranous inflammation, Hemorrhagic inflammation, Allergic inflammation.

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Ulcerative Inflammation

Ulcerative colitis (UC) is a chronic inflammatory and ulcerative disease arising in the colonic mucosa, characterized most often by bloody diarrhea. Extra intestinal symptoms, particularly arthritis, may occur. Long-term risk of colon cancer is high. Diagnosis is by colonoscopy. Treatment is with 5-aminosalicylic acid, corticosteroids, immunomodulators, anticytokines, antibiotics, and occasionally surgery

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Archives of inflammation, Immunogenetics: Open Access, Infectious Diseases & Therapy, Immunome Research, Immunome Research, Cytokine & Growth Factor Reviews, Current opinion on Immunology, Developmental & Comparative Immunology, Human Immunology, Immunology Letters

Proinflammatory Mediators

Chemical mediators of the inflammatory process include a variety of substances originating in the plasma and the cells of uninjured tissue, and possibly from the damaged tissue. The major kinds of mediators are (1) vasoactive amines, such as histamine and serotonin; (2) plasma endopeptidases that comprise three interrelated systems, the kinin system that produces bradykinin, the complement system that produces proteins that interact with antigen--antibody complexes and mediate immunologic injury and inflammation, and the clotting system that increases vascular permeability and chemotactic activity for the leukocytes

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